References

Gunningberg L, Lindholm C, Carlsson M, Sjoden PO. Risk, prevention and treatment of pressure ulcers- -nursing staff knowledge and documentation. Scand J Caring Sci. 2001; 15:(3)257-263 https://doi.org/10.1046/j.1471-6712.2001.00034.x

Shea JD. Pressure sores: classification and management. Clin Orthop Relat Res. 1975; 112:89-100

Nightingale F. Notes on nursing.: Lippincott;

Guttman L. The problem of treatment of pressure sores in spinal paraplegics. Brit J Plast Surg. 1955; 8:196-213 https://doi.org/10.1016/s0007-1226(55)80037-9

Nix DH. Patient assessment and evaluate of healing, 3rd edn. In: Bryant RA, Nix DP. (eds). : Mosby Elsevier; 2007

Lovell ME, Evans JH. A comparison of the spinal board and the vacuum stretcher, spinal stability and interface pressure. Injury. 1994; 25:(3)179-180

Baldwin KM. Transcutaneous oximetry and skin surface temperature as objective measures of pressure ulcer risk. Adv Skin Wound Care,. 2001; 14:(1)26-31

Andrychuk MA. Pressure Ulcers: Causes, Risk Factors, Assessment, and Intervention. Orthop Nurs. 1998; 17:(4)65-81

Langemo DK, Brown G. Skin Fails Too: Acute, Chronic, and End-Stage Skin Failure. Adv Skin Wound Care. 2006; 19:(4)206-211 https://doi.org/10.1097/00129334-200605000-00014

Sprigle S, Linden M, McKenna D Clinical skin temperature measurement to predict incipient pressure ulcers. Advances in Skin & Wound Care. 2001; 14:(3)133-137

Hopkins B, Hanlon M, Yauk S Reducing nosocomial pressure ulcers in an acute care facility.. J Nursing Care. 2000; 14:(3)28-36

Solis L, Hallihan D, Uwiera R Prevention of pressure-induced deep tissue injury using intermittent electrical stimulation. J Appl Physiol. 2007; 102:1992-2001 https://doi.org/10.1152/japplphysiol.01092.2006

Sprigle S, Linden M, Riordan B. Analysis of localized erythema using clinical indicators and spectroscopy. Ostomy/Wound Management. 2003; 49:(3)42-52

Gould LJ, Bohn G, Bryant R Pressure ulcer summit 2018: An interdisciplinary approach to improve our understanding of the risk of pressure induced tissue damage. Wound Repair Regen. 2019; 27:(5)497-508 https://doi.org/10.1111/wrr.12730

Predicting deep tissue injury pressure ulcers: a conceptual model

02 February 2020
Volume 4 · Issue 1

Pressure ulcers (PU) have been described in the literature as early as 2050–100BC and were first identified in Egyptian mummies.1 According to Shea,2 PUs were described in writings of Fabricus, a surgeon from the Netherlands, and Charcot, a French neurologist and pathologist. Indeed, in 1860 Florence Nightingale wrote:

‘… if he has a bedsore, it is generally the fault not of the disease, but of the nursing’.3

In 1955, Guttman4 devised the first classification system for PUs. However, most importantly, a closed PU and four different stages of soft tissue injury was described by Shea.2

The term deep tissue injury (DTI) was introduced as a classification in 2002.5 DTIs appear with intact skin with differing shades of purple hue. This type of PU can deteriorate quickly, within hours to days, depending on the amount of tissue damage present. However, DTIs are difficult to identify in patients with dark skin tone. If you look at Fig 1, there are two pictures of a patient with a light skin tone and a patient with a dark skin tone for comparison. The centre of the DTI reveals black necrotic tissue as the extent of the tissue damage becomes apparent.

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